Hypermetropia
Etiology
Axial
hypermetropia
·
By far the commonest form in which the
total refractive power of the eye is normal but axial length is short
·
About 1mm shortening of the anteroposterior
diameter of the eye results in 3 diopters of hypermetropia
Curvatural
hypermetropia
·
Is the condition in which the curvature of the cornea, lens,
or both is flatter than normal, resulting in a decrease in the refractive power
of the eye
· About 1mm increase in radius of curvature results in 6 diopters of hypermetropia
Index
hypermetropia
·
Occurs due to changes in the refractive
index of the lens in old age. It may also occur in diabetics under treatment
·
Positional hypermetropia
·
Results from posteriorly placed
crystalline lens
·
Absence of crystalline lens
·
Either congenital or acquired
(following surgical removal or posterior dislocation) leads of aphakia
Clinical
types
Simple Hypermetropia
·
Results from normal biological
variants in the development of eyeball
·
It includes axial and curvatural
hypermetropia
Pathological hypermetropia
·
Results due to either congenital or
acquired conditions of the eyeball which are outside the normal biological
variations of the development
Senile hypermetropia / acquired
hypermetropia
occurs in old age due to two causes –
1) curvatural hypermetropia
2) index hypermetropia
Positional hypermetropia may
occur due to posterior subluxation of lens
Aphakia due
to either congenital or acquired (following surgical removal or posterior
dislocation)
Consecutive hypermetropia due
to surgically overcorrected myopia or pseudophakia with undercorrection
Clinical types
Functional Hypermetropia results
from paralysis of accommodation as seen in patients with third nerve palsy and
internal ophthalmoplegia
Components of hypermetropia
Total hypermetropia
Total amount of refractive error which is
estimated after complete cycloplegia with atropine
Latent hypermetropia
·
Implies the amount of hypermetropia
(about 1D) which is normally corrected by inherent tone of ciliary muscle
·
The degree of latent hypermetropia is
high in children and gradually decreases with age
·
Latent hypermetropia disclosed when
refraction is carried after abolishing the tone with atropine
Components
of hypermetropia
Manifest hypermetropia
is the remaining portion of hypermetropia which is not corrected by the ciliary
tone
Facultative
hypermetropia constitutes that part which can be
corrected by the patient’s accommodative effort
Absolute
hypermetropia is the residual part of manifest
hypermetropia which can not be corrected by the patient’s accommodative effort
Symptoms
·
Asymptomatic
·
Asthenopic symptoms
·
Defective vision with asthenopic
symptoms
·
Defective vision with out asthenopic
symptoms
·
The effect of ageing on vision
·
Intermittent sudden blurring of vision
·
Crossed eye sensation
Signs
·
Size of eyeball may appear small
·
Cornea may be slightly smaller than
normal
·
Anterior chamber is comparatively
shallow and angle is narrow
·
Visual acuity varies with the degree
of hypermetropia
·
Fundus examination
Ø small
optic disc which may look more vascular with ill-defined margins
Ø Pseudo
papillitis
Ø Shot
silk appearance
·
A – scan unltrasonograpy may reveal a
short axial length
Complications
·
Recurrent styes, blepharitis or
chalazion may occur. Probably due to infection introduced by repeated rubbing of
eyes
·
Accommodative convergent squint may
develop in childen due to excessive use of accommodation
·
Amblyopia may develop in some cases,
may be anisometropic, strabismic or ametropic
·
Predisposition to develop primary
narrow angle glaucoma
Treatment
·
Total amount of hypermetropia should
always be discovered by performing refraction under complete cycloplegia
·
If the total manifest refractive error
is small, correction is given only if the patient is symptomatic
·
Gradually increase the spherical
correction at 6 months interval till the patient accepts manifest hypermetropia
·
If there is associated exophoria, the
hyperopia should be under corrected by 1 to 2 D
·
In the presence of accommodative
convergent squint, full correction should be given at the first sitting
·
If there is associated amblyopia, full
correction with occlusion therapy should be started
·
It is important to remember that in
children hypermetropia may diminish with the growth of the child. So refraction
should be carried out every six months and if necessary the correction should
be reduced
There are
several optical and surgical treatment options available for hypermetropia:
Optical treatment:
Spectacle: Corrective lenses,
including eyeglasses, are the most common way to correct hypermetropia. The
lenses are designed to bend the light entering the eye so that it focuses
correctly on the retina.
Contact lenses: Like eyeglasses,
contact lenses can also correct hypermetropia. They are placed directly on the
eye and work by changing the way that light enters the eye.
Surgical treatment:
LASIK: Laser-assisted in-situ
keratomileusis (LASIK) is a surgical procedure that uses a laser to reshape the
cornea. This changes the way that light enters the eye, correcting
hypermetropia.
PRK: Photorefractive
keratectomy (PRK) is similar to LASIK but does not involve creating a flap in
the cornea. Instead, the outer layer of the cornea is removed, and a laser is
used to reshape the underlying tissue.
LASEK: Laser epithelial
keratomileusis (LASEK) is similar to PRK but involves preserving a thin layer
of the cornea's outer layer (epithelium) to aid in healing.
Phakic intraocular lenses (IOLs):
Phakic IOLs are artificial lenses that are surgically implanted into the eye.
They are designed to correct refractive errors and can be used to correct
hypermetropia.
Clear lens exchange (CLE): CLE is
a procedure in which the natural lens of the eye is removed and replaced with
an artificial lens that corrects refractive errors. This can be used to correct
hypermetropia, particularly in people who are not good candidates for LASIK or
other laser-based procedures.
Comments
Post a Comment